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(60) utilized next generation sequencing (NGS) and computational analysis to demonstrate that miR-23b-3p, acting as a tumor suppressor, can inhibit tumor Gln metabolism by targeting GLS1, thereby enhancing the sensitivity of liver tumor cells to sorafenib

These glial alterations are thought to interfere with proper neural circuit formation and functions, along with chronic inflammation

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The compound has therefore become a point of interest for its potential role in experimental studies exploring melanocortin receptors, neuroinflammation, and tissue fibrosis

Current prophylactic treatments targeting mitochondrial function and energy metabolism include riboflavin, coenzyme Q10, alpha-lipoic acid, and ketogenic ketones