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doi: 10.1016/0092-8674(93)90509-o
These require unlocking by macrophages, which can release reactive, labile iron into the blood
In chronic or biofilm-associated infections, however, microbial persistence and metabolic cues from the wound microbiome can skew phagocyte polarization toward a dysfunctional state either sustaining a prolonged M1-like inflammatory profile that damages tissue or inducing an immunosuppressed M2-like state that allows microbial survival [42]
Chemosphere 199:1625
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