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Notably, in addition to subcutaneous injections, the first oral Lp(a)-reducing small molecule, Muvalaplin, has also recently achieved a breakthrough, with a Phase I clinical study demonstrating that daily oral doses of Muvalaplin over a 14-day period reduced blood Lp(a) levels by up to 65 per cent [238]

A previous study on the association between KEGG pathways and AD revealed that the pathways most closely related to AD pathology-associated genes or targets of approved AD drugs were not necessarily the most extensively studied, highlighting a disconnect between drug development and mechanism research [3]

The corresponding metabolitegene interaction pairs underlying ASDcontrol differences are summarized in Supplementary Table S3