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Michelle Oravitz: hmm

Notably, Through a feedback mechanism, mTORC1 inhibition may momentarily increase mTORC2 signaling, causing temporary metabolic disruptions during the first phase of treatment (224), which can transiently disrupt metabolism and frequently leads to hyperlipidemia, necessitating concomitant lipid-lowering therapy (237239)

& Titone, D

RB, JB, CA, MS, VS, MT, SD, ZS, KJ, and RD: formal analysis

This suggests that glutathionylation of one of these cysteine residues may be an intermediate toward the formation of the intermolecular disulfide in isoforms possessing both cysteines

Stock V, Laurisch C, Franke J, Dnmez MH, Voss L, Bhmert L, et al