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One of the early events in the development of CAVD is the activation of valvular interstitial cells (VICs), which maintain the structural integrity and function of the valve 33

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Their development, survival, and function are regulated by the colony-stimulating factor 1 receptor, the transcription factor PU.1, and interferon regulatory factor 8 [18]

Further, GLP-1RAs significantly reduce the risk of MI and stroke, but have a neutral effect on hospitalization for heart failure upon 5 available CVOTs [49]