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Rotenone treated in vitro PD model showed impairment of lysosomal functions due to decreased NAD + /NADH and Sirt1 expression ( In a study, extracellular S has been found to upregulate Sirt3 and Sirt5 but downregulate Sirt1 ( Pharmacological activation and inhibition of Sirts have shown protective effects against apoptotic cell death in PD models

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[1] [2] The medication works by mimicking the natural hormone GLP-1, which regulates appetite and food intake by acting on areas of the brain that control hunger

This deficiency can cause serious health issues like fatigue, neurological problems, and blood problems

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