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This process is driven by disrupted GSH homeostasis, resulting from diminished GSH synthesis and impaired activity of two critical antioxidant systems: (1) the cystine/glutamate antiporter xCT, which imports cystine to fuel GSH production and (2) GPX4, which utilizes GSH to convert lipid peroxides into less toxic lipid alcohols (Stockwell et al., 2017

This additional functional domain distinguishes thymosin beta-4 from TB-500 and helps explain the parent molecules wider spectrum of biological activity

doi: 10.1016/j.neubiorev.2024.105728 123 BlattGJFatemiSH

Lenzi, A., Lombardo, F., Gandini, L., Culasso, F., & Dondero, F

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