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In neonates, inherited defects of any of the enzymes of the urea cycle lead to hyperammonemia , which impairs the function of the brain, causing encephalopathy

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INTRODUCTION Cellular antioxidant networks are essential for maintaining redox homeostasis, balancing the suppression of oxidative damage with the controlled transmission of redox signals that shape cell fate decisions

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Ketone ester treatment of the 3xTgAD Alzheimers mice reduced the hippocampal mitochondrial [NAD + ]/[NADH] as indicated by the reduced acetoacetate/beta-hydroxybutyrate that changed from 9.3 to 3.4 (2.7-fold decrease), while there was no significant change in this ratio in the cerebral cortex (Pawlosky et al., 2017)

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