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Cardiomyocytes isolated from mice fed a high-fat/high-sugar diet (HFSD) and subjected to mechanical stretch exhibited significantly increased stiffness (stress/strain) in vitro , correlating with diastolic dysfunction (E/e') observed in vivo

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Although the exact mechanism is not currently known, it is evident that there is a cross-talk between the IGF-1 system, ER and the cognate ER ligand 17-estradiol (E2)